ROLE OF CYTOKINES AND THROMBOPHILIA GENES IN THE ETIOPATHOGENESIS OF ISCHEMIC STROKE AFTER COVID-19

Abstract

It has been found that coronaviruses can lead to an excessive, unregulated
immune response in the host body - cytokine release syndrome. Information is
increasingly being heard that this type of immune response is an integral part of the
development of target organ dysfunction and one of the main factors of morbidity
and mortality. In particular, it can serve as a background in the development of
acute respiratory distress syndrome (ARDS) in patients with COVID-19