THROMBOPHILIA GENES IN THE ETIOPATHOGENESIS OF ISCHEMIC STROKE AFTER COVID-19

Abstract

The emergence of COVID-19 has brought to light a myriad of vascular
complications, including an increased risk of thrombotic events such as ischemic
stroke. Understanding the interplay between thrombophilia genes and the
development of ischemic stroke following COVID-19 infection is crucial for
uncovering potential genetic predispositions and enhancing clinical management
strategies. This article explores the intricate connection between thrombophilia
genetic factors and the etiopathogenesis of ischemic stroke in the context of COVID19. By delving into the molecular mechanisms underlying thrombotic events postCOVID-19 and the impact of genetic variants associated with thrombophilia, this
study sheds light on the complex interplay between viral infections, genetic
susceptibilities, and cerebrovascular complications. The findings presented herein
aim to contribute to a deeper understanding of the pathophysiological pathways
linking thrombophilia genes to ischemic stroke occurrence after COVID-19, paving
the way for tailored approaches in risk assessment, prevention, and treatment
interventions in affected individuals.